Rabbit newborn with frontal encephalocele

 

 

Esther van Praag, Ph.D.

 

 

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It is rare that a newborn rabbit presents with a deformation of the head. There are two possible origins for this issue: either it is hereditary or it is linked to a developmental defect of the fetus. Encephalocele is a condition that arises from an abnormal development of the neural tube during the early stages of gestation. This defect is associated with an autosomal recessive gene that is expressed at the level of the spinal cord (spina bifida) or the skull (encephalocele).

http://ag.ansc.purdue.edu/nielsen/www495/slides/spina.html

Rabbit fetus that presents an open severe spina bifida. Note the absence of vertebra in place of the vertebral column. This fetus is not viable.

At the skull level, this defect impedes the proper closure of the skull bones and the skull. This incomplete closing is located at the sagittal level, between the forehead and the nostrils. A protrusion, also known as a hernia of the skull, contains part of the brains and sometimes the membranes that surround and protect the brain and spinal cord.

At the time of writing this article, Geelen's 1974 study is the only one to provide a detailed account of hydrocephalus in a rabbit. In this case, the three-week-old rabbit was the result of inbreeding.

This one-day-old newborn rabbit is one of eleven offspring of a female Belgian Beard rabbit, an older breed that became almost extinct. Although it was alive and active, yet, the decision was made to humanely euthanize the newborn, as its prospects for survival in a 11-member nest were dismal and further suffering would be inflicted upon it.

Michel Gruaz

Belgian Beard rabbit newborn aged 1 day that suffer a frontal encephalocele and detail of the head.

Acknowledgement

I would like to express my sincere gratitude to Michel Gruaz of Switzerland for sharing information regarding this rare defect in a newborn rabbit.

Further information

Geelen JAG. A case of hydrocephalus and meningo encephalocele in a rabbit caused by aqueductal malformation. Laboratory Animals (London) 1974 : 167-176.

 

 

 

 

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